Neuromuscular Junction Disorders: Handbook of Clinical Neurology, cartea 91
Editat de A. G. Engelen Limba Engleză Hardback – 3 aug 2008
New observations into the immunopathogenesis of myasthenia gravis, and the molecular mechanisms underlying the links between activity and patterns of gene expression, particularly in muscle, are also explored, along with novel information on the understanding of the pathophysiology of the heterogeneous group of peripheral nerve hyperexcitability syndromes.
Each of these valuable insights, and their accompanying explanations, provides a framework on which new therapeutic strategies can be built to aid in the enhancement and restoration of normal function in neuromuscular junction disorders.
- New insights into the study of neuromuscular junction disorders, including rapid advancements in the field
- Comprehensive information on the neuromuscular junction, and a detailed examination of myasthenia gravis and other disorders
- A new framework upon which to build therapeutic strategies that will enhance the restoration of normal function in neuromuscular disorders
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Specificații
ISBN-13: 9780444520081
ISBN-10: 0444520082
Pagini: 480
Ilustrații: Approx. 180 illustrations (50 in full color)
Dimensiuni: 192 x 262 x 28 mm
Greutate: 1.5 kg
Ediția:New.
Editura: ELSEVIER SCIENCE
Seria Handbook of Clinical Neurology
ISBN-10: 0444520082
Pagini: 480
Ilustrații: Approx. 180 illustrations (50 in full color)
Dimensiuni: 192 x 262 x 28 mm
Greutate: 1.5 kg
Ediția:New.
Editura: ELSEVIER SCIENCE
Seria Handbook of Clinical Neurology
Public țintă
NeurologistsNeuroscience research workers
Cuprins
The most vulnerable synapse: historic aspects of neuromuscular junction disorders. Reliability of neuromuscular transmission and how it is maintained. The neuromuscular junction. Electromyographic aspects of neuromuscular junction disorders. The immunopathogenesis of myasthenia gravis. Autoantibodies in different forms of myasthenia gravis and in the Lambert-Eaton syndrome. Clinical features of myasthenia gravis. Therapy of myasthenia gravis. The Lambert-Eaton myasthenic syndrome. Congenital myasthenic syndromes. Botulism. Neurotoxicology of neuromuscular transmission. Organophosphate and carbamate poisoning. Peripheral nerve hyperexcitability and the neuromuscular junction.